产品名称:NCI-N87-EGFP-LUC人胃,癌细胞-绿色荧光蛋白-荧光素酶标记(STR鉴定正确)、NCI-N87-EGFP-LUC细胞、NCI-N87-EGFP-LUC人胃,癌细胞-绿色荧光蛋白-荧光素酶标记(STR鉴定正确)、NCI-N87-EGFP-LUC细胞、NCI-N87-EGFP-LUC人胃,癌细胞-绿色荧光蛋白-荧光素酶标记(STR鉴定正确)、NCI-N87-EGFP-LUC细胞、NCI-N87-EGFP-LUC人胃,癌细胞-绿色荧光蛋白-荧光素酶标记(STR鉴定正确)、NCI-N87-EGFP-LUC细胞、NCI-N87-EGFP-LUC人胃,癌细胞-绿色荧光蛋白-荧光素酶标记(STR鉴定正确)、NCI-N87-EGFP-LUC细胞

intracellular pathogens into class II molecules, further implicating autophagy in the

regulation of the immune response [ 17]. Several excellent reviews have been recently

published covering the different roles that autophagy plays in the innate and the adaptive immune response [ 18, 19] and we will not cover most of those topics here. Instead this    review will focus on our current knowledge of how different forms of autophagy control specific aspectsofT-cell biology.